This trial for systematic reviews or meta-analyses was registered at PROSPERO as CRD42024525197 (https://www.crd.york.ac.uk/prospero/display_record.php?RecordID=525197).
Sorry since when did the monkeys before us got atherosclerosis? They were dead before the diet came into effect. We live much longer, where we need to keep the plaque development much lower than they had. And don’t tell me that your butter steak doest cause a rise in LDL if baseline LDL is <80 or that high LDL is not causal to atherosclerosis.
LDL is not a disease, its necessary for human life. Damaged (Oxidated, and Glycated) LDL is a indicator of significant cardiovascular problem, but undamaged LDL is not a disease. If someone has elevated LDL and they are not eating a sugar heavy diet, they should get plaque imaging to see what their actual atherosclerotic risk is.
Sorry since when did the monkeys before us got atherosclerosis? They were dead before the diet came into effect. We live much longer, where we need to keep the plaque development much lower than they had. And don’t tell me that your butter steak doest cause a rise in LDL if baseline LDL is <80 or that high LDL is not causal to atherosclerosis.
LDL is not a disease, its necessary for human life. Damaged (Oxidated, and Glycated) LDL is a indicator of significant cardiovascular problem, but undamaged LDL is not a disease. If someone has elevated LDL and they are not eating a sugar heavy diet, they should get plaque imaging to see what their actual atherosclerotic risk is.